Full text not available from this repository.
Although the nature of the infectious agent causing prion diseases is still debated, several of its molecular characteristics have been clarified in remarkable detail. The transmissibility of bovine spongiform encephalopathy to humans dramatically highlights the need for research focused at interference with prion replication and spread, and at prevention of brain damage. Precondition to achieving these goals is a thorough understanding of prion biology, and in particular of its protein chemistry.
|Item Type:||Journal Article, refereed|
|Communities & Collections:||04 Faculty of Medicine > University Hospital Zurich > Institute of Neuropathology|
|DDC:||570 Life sciences; biology|
610 Medicine & health
|Date:||1 July 1998|
|Deposited On:||11 Feb 2008 12:25|
|Last Modified:||27 Nov 2013 17:29|
|Citations:||Web of Science®. Times Cited: 18|
Users (please log in): suggest update or correction for this item
Repository Staff Only: item control page