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Increased brain beta-amyloid load, phosphorylated tau, and risk of Alzheimer disease associated with an intronic CYP46 polymorphism


Papassotiropoulos, A; Streffer, J R; Tsolaki, M; Schmid, S; Thal, D; Nicosia, F; Iakovidou, V; Maddalena, A; Lütjohann, D; Ghebremedhin, E; Hegi, T; Pasch, T; Träxler, M; Brühl, A; Benussi, L; Binetti, G; Braak, H; Nitsch, R M; Hock, C (2003). Increased brain beta-amyloid load, phosphorylated tau, and risk of Alzheimer disease associated with an intronic CYP46 polymorphism. Archives of Neurology, 60(1):29-35.

Abstract

CYP46 influences brain beta-amyloid load, cerebrospinal fluid levels of beta-amyloid peptides and phosphorylated tau, and the genetic risk of late-onset sporadic AD.

CYP46 influences brain beta-amyloid load, cerebrospinal fluid levels of beta-amyloid peptides and phosphorylated tau, and the genetic risk of late-onset sporadic AD.

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Additional indexing

Item Type:Journal Article, refereed, original work
Communities & Collections:04 Faculty of Medicine > Institute for Regenerative Medicine (IREM)
Dewey Decimal Classification:610 Medicine & health
Language:English
Date:2003
Deposited On:08 Sep 2011 08:28
Last Modified:16 Aug 2016 10:15
Publisher:American Medical Association
ISSN:0003-9942
Free access at:Official URL. An embargo period may apply.
Publisher DOI:https://doi.org/10.1001/archneur.60.1.29
Official URL:http://archneur.ama-assn.org/cgi/content/full/60/1/29
PubMed ID:12533085

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