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Influenza A virus lures autophagic protein LC3 to budding sites


Münz, Christian (2014). Influenza A virus lures autophagic protein LC3 to budding sites. Cell Host & Microbe, 15(2):130-131.

Abstract

Autophagy, a cellular degradation pathway, can restrict or assist viral replication. In this issue of Cell Host & Microbe, Beale et al. (2014) report that the influenza virus matrix protein 2 binds to the essential autophagy protein LC3 to presumably transport LC3-conjugated membranes to the cell surface for budding of stable viruses.

Abstract

Autophagy, a cellular degradation pathway, can restrict or assist viral replication. In this issue of Cell Host & Microbe, Beale et al. (2014) report that the influenza virus matrix protein 2 binds to the essential autophagy protein LC3 to presumably transport LC3-conjugated membranes to the cell surface for budding of stable viruses.

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Additional indexing

Item Type:Journal Article, refereed, further contribution
Communities & Collections:04 Faculty of Medicine > Institute of Experimental Immunology
Dewey Decimal Classification:570 Life sciences; biology
610 Medicine & health
Language:English
Date:12 February 2014
Deposited On:09 Dec 2014 15:32
Last Modified:08 Dec 2017 08:44
Publisher:Cell Press (Elsevier)
ISSN:1931-3128
Free access at:Publisher DOI. An embargo period may apply.
Publisher DOI:https://doi.org/10.1016/j.chom.2014.01.014
PubMed ID:24528859

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