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The lectin-like domain of tumor necrosis factor improves lung function after rat lung transplantation-potential role for a reduction in reactive oxygen species generation


Hamacher, J; Stammberger, U; Roux, J; Kumar, S; Yang, G; Xiong, C; Schmid, R A; Fakin, R M; Chakraborty, T; Hossain, H M D; Pittet, J F; Wendel, A; Black, S M; Lucas, R (2010). The lectin-like domain of tumor necrosis factor improves lung function after rat lung transplantation-potential role for a reduction in reactive oxygen species generation. Critical Care Medicine, 38(3):871-878.

Abstract

These data demonstrate that the TIP peptide significantly improves lung function after lung transplantation in the rat, in part, by reducing neutrophil content and reactive oxygen species generation. These studies suggest that the TIP peptide is a potential therapeutic agent against the ischemia reperfusion injury associated with lung transplantation.

Abstract

These data demonstrate that the TIP peptide significantly improves lung function after lung transplantation in the rat, in part, by reducing neutrophil content and reactive oxygen species generation. These studies suggest that the TIP peptide is a potential therapeutic agent against the ischemia reperfusion injury associated with lung transplantation.

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21 citations in Web of Science®
23 citations in Scopus®
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Additional indexing

Item Type:Journal Article, refereed, original work
Communities & Collections:04 Faculty of Medicine > University Hospital Zurich > Clinic for Reconstructive Surgery
Dewey Decimal Classification:610 Medicine & health
Language:English
Date:2010
Deposited On:25 Mar 2011 13:40
Last Modified:05 Apr 2016 14:53
Publisher:Lippincott Wiliams & Wilkins
ISSN:0090-3493
Publisher DOI:https://doi.org/10.1097/CCM.0b013e3181cdf725
PubMed ID:20081530

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