Rossi, F; Khanduja, J S; Bortoluzzi, A; Houghton, J; Sander, P; Güthlein, C; Davis, E O; Springer, B; Böttger, E C; Relini, A; Penco, A; Muniyappa, K; Rizzi, M (2011). The biological and structural characterization of Mycobacterium tuberculosis UvrA provides novel insights into its mechanism of action. Nucleic Acids Research, 39(16):7316-7328.
Williams, A; Güthlein, C; Beresford, N; Böttger, E C; Springer, B; Davis, E O (2011). UvrD2 is essential in mycobacterium tuberculosis, but its helicase activity is not required. Journal of Bacteriology, 193(17):4487-4494.
Güthlein, C; Wanner, R M; Sander, P; Davis, E O; Bosshard, M; Jiricny, J; Böttger, E C; Springer, B (2009). Characterisation of the mycobacterial NER system reveals novel functions of uvrD1 helicase. Journal of Bacteriology, 191(2):555-562.
Rand, L; Hinds, J; Springer, B; Sander, P; Buxton, R S; Davis, E O (2003). The majority of inducible DNA repair genes in Mycobacterium tuberculosis are induced independently of RecA. Molecular Microbiology, 50(3):1031-1042.
Davis, E O; Springer, B; Gopaul, K K; Papavinasasundaram, K G; Sander, P; Böttger, E C (2002). DNA damage induction of recA in Mycobacterium tuberculosis independently of RecA and LexA. Molecular Microbiology, 46(3):791-800.