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Number of items: 11.

Schüpbach, J; Günthard, H F; Joos, B; Fischer, M B; Böni, J; Tomasik, Z; Yerly, S; Perrin, L; Battegay, M; Furrer, H; Vernazza, P; Bernasconi, E; Hirschel, B (2005). HIV-1 p24 may persist during long-term highly active antiretroviral therapy, increases little during short treatment breaks, and its rebound after treatment stop correlates with CD4(+) T cell loss. JAIDS Journal of Acquired Immune Deficiency Syndromes, 40(3):250-256.

Aguzzi, A; Brandner, S; Fischer, M B; Furukawa, H; Glatzel, M; Hawkins, C; Heppner, F L; Montrasio, F; Navarro, B; Parizek, P; Pekarik, V; Prinz, M; Raeber, A J; Röckl, C; Klein, M A (2001). Spongiform encephalopathies: insights from transgenic models. Advances in Virus Research, 56:313-352.

Fischer, M B; Roeckl, C; Parizek, P; Schwarz, H P; Aguzzi, A (2000). Binding of disease-associated prion protein to plasminogen. Nature, 408(6811):479-483.

Flechsig, E; Shmerling, D; Hegyi, I; Raeber, A J; Fischer, M B; Cozzio, A; von Mering, C; Aguzzi, A; Weissmann, C (2000). Prion protein devoid of the octapeptide repeat region restores susceptibility to scrapie in PrP knockout mice. Neuron, 27(2):399-408.

Raeber, A J; Sailer, A; Hegyi, I; Klein, M A; Rülicke, T; Fischer, M B; Brandner, S; Aguzzi, A; Weissmann, C (1999). Ectopic expression of prion protein (PrP) in T lymphocytes or hepatocytes of PrP knockout mice is insufficient to sustain prion replication. Proceedings of the National Academy of Sciences of the United States of America (PNAS), 96(7):3987-3992.

Raeber, A J; Brandner, S; Klein, M A; Benninger, Y; Musahl, C; Frigg, R; Roeckl, C; Fischer, M B; Weissmann, C; Aguzzi, A (1998). Transgenic and knockout mice in research on prion diseases. Brain Pathology, 8(4):715-733.

Shmerling, D; Hegyi, I; Fischer, M B; Blättler, T; Brandner, S; Götz, J; Rülicke, T; Flechsig, E; Cozzio, A; von Mering, C; Hangartner, C; Aguzzi, A; Weissmann, C (1998). Expression of amino-terminally truncated PrP in the mouse leading to ataxia and specific cerebellar lesions. Cell, 93(2):203-214.

Aguzzi, A; D'Angelo, M G; Klein, M A; Frigg, R; Fischer, M B; Raeber, A J; Röckl, C; Musahl, C; Hegyi, I; Brandner, S (1998). [The new research frontier on prion disease.]. Pathologica, 90(6):743-752.

Brandner, S; Raeber, A; Sailer, A; Blättler, T; Fischer, M B; Weissmann, C; Aguzzi, A (1996). Normal host prion protein (PrPC) is required for scrapie spread within the central nervous system. Proceedings of the National Academy of Sciences of the United States of America (PNAS), 93(23):13148-13151.

Fischer, M B; Rülicke, T; Raeber, A; Sailer, A; Moser, M; Oesch, B; Brandner, S; Aguzzi, A; Weissmann, C (1996). Prion protein (PrP) with amino-proximal deletions restoring susceptibility of PrP knockout mice to scrapie. The EMBO Journal, 15(6):1255-1264.

Brandner, S; Isenmann, S; Raeber, A; Fischer, M B; Sailer, A; Kobayashi, Y; Marino, S; Weissmann, C; Aguzzi, A (1996). Normal host prion protein necessary for scrapie-induced neurotoxicity. Nature, 379(6563):339-343.

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