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Number of items: 12.

Derungs, R; Camici, G G; Spescha, R D; Welt, T; Tackenberg, C; Späni, C; Wirth, F; Grimm, A; Eckert, A; Nitsch, R M; Kulic, L (2017). Genetic ablation of the p66(Shc) adaptor protein reverses cognitive deficits and improves mitochondrial function in an APP transgenic mouse model of Alzheimer's disease. Molecular Psychiatry, 22(4):605-614.

Ferretti, M T; Merlini, M; Späni, C; Gericke, C; Schweizer, N; Enzmann, G; Engelhardt, B; Kulic, L; Suter, T; Nitsch, R M (2016). T-cell brain infiltration and immature antigen-presenting cells in transgenic models of Alzheimer's disease-like cerebral amyloidosis. Brain, Behavior, and Immunity, 54:211-225.

Spescha, R D; Klohs, J; Semerano, A; Giacalone, G; Derungs, R S; Reiner, M F; Rodriguez Gutierrez, D; Mendez-Carmona, N; Glanzmann, M; Savarese, G; Kränkel, N; Akhmedov, A; Keller, S; Mocharla, P; Kaufmann, M R; Wenger, R H; Vogel, J; Kulic, L; Nitsch, R M; Beer, J H; Peruzzotti-Jametti, L; Sessa, Maria; Lüscher, Thomas F; Camici, G G (2015). Post-ischaemic silencing of p66Shc reduces ischaemia/reperfusion brain injury and its expression correlates to clinical outcome in stroke. European Heart Journal, 36(25):1590-1600.

Santuccione, A C; Merlini, M; Shetty, A; Tackenberg, C; Bali, J; Ferretti, M T; McAfoose, J; Kulic, L; Bernreuther, C; Welt, T; Grimm, J; Glatzel, M; Rajendran, L; Hock, C; Nitsch, R M (2013). Active vaccination with ankyrin G reduces β-amyloid pathology in APP transgenic mice. Molecular Psychiatry, 18(3):358-368.

Farkas, M; Keskitalo, S; Smith, D E C; Bain, N; Semmler, A; Ineichen, B; Smulders, Y; Blom, H; Kulic, L; Linnebank, M (2013). Hyperhomocysteinemia in Alzheimer's disease: The hen and the egg? Journal of Alzheimer's disease : JAD, 33(4):1097-1104.

Kulic, L; McAfoose, J; Welt, T; Tackenberg, C; Späni, C; Wirth, F; Finder, V; Konietzko, U; Giese, M; Eckert, A; Noriaki, K; Shimizu, T; Murakami, K; Irie, K; Rasool, S; Glabe, C; Hock, C; Nitsch, R M (2012). Early accumulation of intracellular fibrillar oligomers and late congophilic amyloid angiopathy in mice expressing the Osaka intra-Aβ APP mutation. Translational Psychiatry, 2:e183.

Cattepoel, S; Hanenberg, M; Kulic, L; Nitsch, R M (2011). Chronic intranasal treatment with an anti-Aβ(30-42) scFv antibody ameliorates amyloid pathology in a transgenic mouse model of Alzheimer's disease. PLoS ONE, 6(4):e18296.

Kulic, L; Wollmer, M A; Rhein, V; Pagani, L; Kuehnle, K; Cattepoel, S; Tracy, J; Eckert, A; Nitsch, R M (2011). Combined expression of tau and the Harlequin mouse mutation leads to increased mitochondrial dysfunction, tau pathology and neurodegeneration. Neurobiology of Aging, 32(10):1827-1838.

Linnebank, M; Popp, J; Smulders, Y; Smith, D; Semmler, A; Farkas, M; Kulic, L; Cvetanovska, G; Blom, H; Stoffel-Wagner, B; Kölsch, H; Weller, M; Jessen, F (2010). S-Adenosylmethionine is decreased in the cerebrospinal fluid of patients with Alzheimer's disease. Neurodegenerative Diseases, 7(6):373-378.

Kuehnle, K; Ledesma, M D; Kalvodova, L; Smith, A E; Crameri, A; Skaanes-Brunner, F; Thelen, K M; Kulic, L; Lütjohann, D; Heppner, F L; Nitsch, R M; Mohajeri, M H (2009). Age-dependent increase in desmosterol restores DRM formation and membrane-related functions in cholesterol-free DHCR24(-/-) mice. Neurochemical Research (34, 1167-82). Neurochemical Research, 34(8):1522.

Kuehnle, K; Crameri, A; Kälin, R E; Luciani, P; Benvenuti, S; Peri, A; Ratti, F; Rodolfo, M; Kulic, L; Heppner, F L; Nitsch, R M; Mohajeri, M H (2008). Prosurvival effect of DHCR24/Seladin-1 in acute and chronic responses to oxidative stress. Molecular and Cellular Biology, 28(2):539-550.

Kulic, L; Kurosinski, P; Chen, F; Tracy, J; Mohajeri, M H; Li, H; Nitsch, R M; Götz, J (2006). Active immunization trial in Abeta42-injected P301L tau transgenic mice. Neurobiology of Disease, 22(1):50-56.

This list was generated on Wed Oct 18 00:38:03 2017 CEST.