Number of items: 6.
Oertli, M; Engler, D B; Kohler, E; Koch, M; Meyer, T F; Müller, A (2011). MicroRNA-155 is essential for the T cell-mediated control of Helicobacter pylori infection and for the induction of chronic gastritis and colitis. Journal of Immunology, 187(7):3578-3586.
Gressmann, H; Linz, B; Ghai, R; Pleissner, K P; Schlapbach, R; Yamaoka, Y; Kraft, C; Suerbaum, S; Meyer, T F; Achtman, M (2005). Gain and loss of multiple genes during the evolution of Helicobacter pylori. PLoS Genetics, 1(4):e43.
Müller, A; Rassow, J; Grimm, J; Machuy, N; Meyer, T F; Rudel, T (2002). VDAC and the bacterial porin PorB of Neisseria gonorrhoeae share mitochondrial import pathways. EMBO Journal, 21(8):1916-1929.
Rajalingam, K; Al-Younes, H; Müller, A; Meyer, T F; Szczepek, A J; Rudel, T (2001). Epithelial cells infected with Chlamydophila pneumoniae (Chlamydia pneumoniae) are resistant to apoptosis. Infection and Immunity, 69(12):7880-7888.
Müller, A; Günther, D; Brinkmann, V; Hurwitz, R; Meyer, T F; Rudel, T (2000). Targeting of the pro-apoptotic VDAC-like porin (PorB) of Neisseria gonorrhoeae to mitochondria of infected cells. EMBO Journal, 19(20):5332-543.
Müller, A; Günther, D; Düx, F; Naumann, M; Meyer, T F; Rudel, T (1999). Neisserial porin (PorB) causes rapid calcium influx in target cells and induces apoptosis by the activation of cysteine proteases. EMBO Journal, 18(2):339-352.
This list was generated on Tue Jul 25 11:19:04 2017 CEST.
