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Number of items: 16.

Potes, C S; Boyle, C N; Wookey, P J; Riediger, T; Lutz, T A (2012). Involvement of the extracellular-signal regulated kinase 1/2 signaling pathway in amylin's eating inhibitory effect. American Journal of Physiology. Regulatory, Integrative and Comparative Physiology, 302(3):R340-51.

Potes, C S; Lutz, T A; Riediger, T (2010). Identification of central projections from amylin-activated neurons to the lateral hypothalamus. Brain Research, 1334:31-44.

Riediger, T; Eisele, N; Scheel, C; Lutz, T A (2010). Effects of glucagon-like peptide 1 and oxyntomodulin on neuronal activity of ghrelin-sensitive neurons in the hypothalamic arcuate nucleus. American Journal of Physiology. Regulatory, Integrative and Comparative Physiology, 298(4):R1061-R1067.

Riediger, T; Cordani, C; Potes, C S; Lutz, T A (2010). Involvement of nitric oxide in lipopolysaccharide induced anorexia. Pharmacology Biochemistry and Behavior, 97(1):112-120.

Potes, C S; Turek, V F; Cole, R L; Vu, C; Roland, B L; Roth, J D; Riediger, T; Lutz, T A (2010). Noradrenergic neurons of the area postrema mediate amylin's hypophagic action. American Journal of Physiology. Regulatory, Integrative and Comparative Physiology, 299(2):R623-R631.

Becskei, C; Lutz, T A; Riediger, T (2010). Reduced fasting-induced activation of hypothalamic arcuate neurons is associated with hyperleptinemia and increased leptin sensitivity in obese mice. American Journal of Physiology. Regulatory, Integrative and Comparative Physiology, 299(2):632-641.

Becskei, C; Lutz, T A; Riediger, T (2009). Blunted fasting-induced hypothalamic activation and refeeding hyperphagia in late-onset obesity. Neuroendocrinology, 90(4):371-382.

Becskei, C; Lutz, T A; Riediger, T (2009). Diet-derived nutrients mediate the inhibition of hypothalamic NPY neurons in the arcuate nucleus of mice during refeeding. American Journal of Physiology. Regulatory, Integrative and Comparative Physiology, 297(1):100-110.

Potes, C S; Riediger, T; Lutz, T A (2009). Noradrenergic neurons of the area postrema mediate amylin's anorectic action. Appetite, 52(3):853.

Riediger, T; Michel, S; Forster, K; Lutz, T A (2009). The ability of amylin to reduce eating depends on the protein content of the diet. Appetite, 52(3):854.

The central signaling pathways of the anorectic hormone amylin: a neuroanatomical study. Edited by: Potes, C S; Lutz, T A; Riediger, T (2008). Paris: Society for the Study of Ingestive Behavior SSIB.

Becskei, C; Riediger, T; Hernádfalvy, N; Arsenijevic, D; Lutz, T A; Langhans, W (2008). Inhibitory effects of lipopolysaccharide on hypothalamic nuclei implicated in the control of food intake. Brain, Behavior, and Immunity, 22(1):56-64.

Becskei, C; Lutz, T A; Riediger, T (2008). Glucose reverses fasting-induced activation in the arcuate nucleus of mice. NeuroReport, 19(1):105-109.

Becskei, C; Bilik, K U; Klussmann, S; Jarosch, F; Lutz, T A; Riediger, T (2008). The anti-ghrelin Spiegelmer NOX-B11-3 blocks ghrelin- but not fasting-induced neuronal activation in the hypothalamic arcuate nucleus. Journal of Neuroendocrinology, 20(1):85-92.

Bilik, K U; Ergüven, E; Klussmann, S; Jarosch, F; Wielinga, P Y; Lutz, T A; Riediger, T (2007). In-vitro and in-vivo antagonistic action of an anti-amylin Spiegelmer. NeuroReport, 18(17):1855-1859.

Becskei, C; Grabler, V; Edwards, G L; Riediger, T; Lutz, T A (2007). Lesion of the lateral parabrachial nucleus attenuates the anorectic effect of peripheral amylin and CCK. Brain Research, 1162:76-84.

This list was generated on Thu Nov 23 18:20:54 2017 CET.