Publication: Persistence of Epstein-Barr virus in self-reactive memory B cells
Persistence of Epstein-Barr virus in self-reactive memory B cells
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Tracy, S. I., Kakalacheva, K., Lünemann, J. D., Luzuriaga, K., Middeldorp, J., & Thorley-Lawson, D. A. (2012). Persistence of Epstein-Barr virus in self-reactive memory B cells. Journal of Virology, 86(22), 12330–12340. https://doi.org/10.1128/JVI.01699-12
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Epstein-Barr virus infection has been epidemiologically associated with the development of multiple autoimmune diseases, particularly systemic lupus erythematosus and multiple sclerosis. Currently, there is no known mechanism that can account for these associations. The germinal-center (GC) model of EBV infection and persistence proposes that EBV gains access to the memory B cell compartment via GC reactions by driving infected cells to differentiate using the virus-encoded LMP1 and LMP2a proteins, which act as functional homologues o
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- Tracy, Sean I
- Kakalacheva, Kristina
- Lünemann, Jan D
- Luzuriaga, Katherine
- Middeldorp, Jaap
- Thorley-Lawson, David A
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Tracy, S. I., Kakalacheva, K., Lünemann, J. D., Luzuriaga, K., Middeldorp, J., & Thorley-Lawson, D. A. (2012). Persistence of Epstein-Barr virus in self-reactive memory B cells. Journal of Virology, 86(22), 12330–12340. https://doi.org/10.1128/JVI.01699-12