Publication: Aldosterone-dependent and -independent regulation of the epithelial sodium channel (ENaC) in mouse distal nephron
Aldosterone-dependent and -independent regulation of the epithelial sodium channel (ENaC) in mouse distal nephron
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Nesterov, V., Dahlmann, A., Krueger, B., Bertog, M., Loffing, J., & Korbmacher, C. (2012). Aldosterone-dependent and -independent regulation of the epithelial sodium channel (ENaC) in mouse distal nephron. American Journal of Physiology : Renal Physiology, F1289–F1299. https://doi.org/10.1152/ajprenal.00247.2012
Abstract
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Abstract
Aldosterone is thought to be the main hormone to stimulate the epithelial sodium channel (ENaC) in the aldosterone-sensitive distal nephron (ASDN) comprising the late distal convoluted tubule (DCT2), the connecting tubule (CNT) and the entire collecting duct (CD). There is immunohistochemical evidence for an axial gradient of ENaC expression along the ASDN with highest expression in the DCT2 and CNT. However, most of our knowledge about renal ENaC function stems from studies in the cortical collecting duct (CCD). Here we investigated
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- Nesterov, Viatcheslav
- Dahlmann, Anke
- Krueger, Bettina
- Bertog, Marko
- Loffing, Johannes
- Korbmacher, Christoph
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Nesterov, V., Dahlmann, A., Krueger, B., Bertog, M., Loffing, J., & Korbmacher, C. (2012). Aldosterone-dependent and -independent regulation of the epithelial sodium channel (ENaC) in mouse distal nephron. American Journal of Physiology : Renal Physiology, F1289–F1299. https://doi.org/10.1152/ajprenal.00247.2012
