Publication: Rapid dephosphorylation of the renal sodium chloride cotransporter in response to oral potassium intake in mice
Rapid dephosphorylation of the renal sodium chloride cotransporter in response to oral potassium intake in mice
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Sorensen, M. V., Grossmann, S., Roesinger, M., Gresko, N., Todkar, A. P., Barmettler, G., Ziegler, U., Odermatt, A., Loffing-Cueni, D., & Loffing, J. (2013). Rapid dephosphorylation of the renal sodium chloride cotransporter in response to oral potassium intake in mice. Kidney International, 83(5), 811–824. https://doi.org/10.1038/ki.2013.14
Abstract
Abstract
Abstract
A dietary potassium load induces a rapid kaliuresis and natriuresis, which may occur even before plasma potassium and aldosterone (aldo) levels increase. Here we sought to gain insight into underlying molecular mechanisms contributing to this response. After gastric gavage of 2% potassium, the plasma potassium concentrations rose rapidly (0.25 h), followed by a significant rise of plasma aldo (0.5 h) in mice. Enhanced urinary potassium and sodium excretion was detectable as early as spot urines could be collected (about 0.5 h). The fu
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Creators (Authors)
- Sorensen, Mads V
- Grossmann, Solveig
- Roesinger, Marian
- Gresko, Nikolay
- Todkar, Abhijeet P
- Barmettler, Gery
- Ziegler, Urs
- Odermatt, Alex
- Loffing-Cueni, Dominique
- Loffing, Johannes
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Citations
Sorensen, M. V., Grossmann, S., Roesinger, M., Gresko, N., Todkar, A. P., Barmettler, G., Ziegler, U., Odermatt, A., Loffing-Cueni, D., & Loffing, J. (2013). Rapid dephosphorylation of the renal sodium chloride cotransporter in response to oral potassium intake in mice. Kidney International, 83(5), 811–824. https://doi.org/10.1038/ki.2013.14
