Publication: Hindbrain noradrenergic input to the hypothalamic PVN mediates the activation of oxytocinergic neurons induced by the satiety factor oleoylethanolamide
Hindbrain noradrenergic input to the hypothalamic PVN mediates the activation of oxytocinergic neurons induced by the satiety factor oleoylethanolamide
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Romano, A., Potes, C. S., Tempesta, B., Cassano, T., Cuomo, V., Lutz, T., & Gaetani, S. (2013). Hindbrain noradrenergic input to the hypothalamic PVN mediates the activation of oxytocinergic neurons induced by the satiety factor oleoylethanolamide. American Journal of Physiology. Endocrinology and Metabolism, 305(10), E1266–E1273. https://doi.org/10.1152/ajpendo.00411.2013
Abstract
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Abstract
Oleoylethanolamide (OEA) is a gut-derived endogenous lipid that stimulates vagal fibers to induce satiety. Our previous work has shown that peripherally administered OEA activates c-fos transcription in the nucleus of the solitary tract (NST) and in the paraventricular nucleus (PVN), where it enhances oxytocin (OXY) expression. The anorexigenic action of OEA is prevented by the intracerebroventricular administration of a selective OXY receptor antagonist, suggesting a necessary role of OXYergic mediation of OEA's effect. The NST is th
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- Romano, Adele
- Potes, Catarina Soares
- Tempesta, Bianca
- Cassano, Tommaso
- Cuomo, Vincenzo
- Lutz, Thomas
- Gaetani, Silvana
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Romano, A., Potes, C. S., Tempesta, B., Cassano, T., Cuomo, V., Lutz, T., & Gaetani, S. (2013). Hindbrain noradrenergic input to the hypothalamic PVN mediates the activation of oxytocinergic neurons induced by the satiety factor oleoylethanolamide. American Journal of Physiology. Endocrinology and Metabolism, 305(10), E1266–E1273. https://doi.org/10.1152/ajpendo.00411.2013
