Publication: Metabolic and neurogenic effects of amylin in the rodent Area postrema
Metabolic and neurogenic effects of amylin in the rodent Area postrema
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Liberini, C. (2016). Metabolic and neurogenic effects of amylin in the rodent Area postrema. (Dissertation, University of Zurich) https://doi.org/10.5167/uzh-131688
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Amylin, also known as islet amyloid polypeptide, is co-secreted with insulin by pancreatic β-cells in response to nutrient stimuli. Amylin reduces food intake and body weight and also acts as adiposity signal to control energy expenditure (EE). Circulating amylin acts centrally by primarily activating neurons of the area postrema (AP), a circumventricular organ (CVO) located in the hindbrain. Amylin receptors (AMYs) are dimers of the calcitonin-like receptor isoform a (CTRa) and of one member of the receptor activity modifying protein
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Liberini, C. (2016). Metabolic and neurogenic effects of amylin in the rodent Area postrema. (Dissertation, University of Zurich) https://doi.org/10.5167/uzh-131688
