Maintenance work on the ZORA database servers is scheduled for Thursday 25th July 2019 from 19:00-20:00. During this period ZORA and JDB will not be available. Thank you for your understanding.
Rossi, F; Khanduja, J S; Bortoluzzi, A; Houghton, J; Sander, P; Güthlein, C; Davis, E O; Springer, B; Böttger, E C; Relini, A; Penco, A; Muniyappa, K; Rizzi, M (2011). The biological and structural characterization of Mycobacterium tuberculosis UvrA provides novel insights into its mechanism of action. Nucleic Acids Research, 39(16):7316-7328.
Williams, A; Güthlein, C; Beresford, N; Böttger, E C; Springer, B; Davis, E O (2011). UvrD2 is essential in mycobacterium tuberculosis, but its helicase activity is not required. Journal of Bacteriology, 193(17):4487-4494.
Güthlein, C; Wanner, R M; Sander, P; Davis, E O; Bosshard, M; Jiricny, J; Böttger, E C; Springer, B (2009). Characterisation of the mycobacterial NER system reveals novel functions of uvrD1 helicase. Journal of Bacteriology, 191(2):555-562.
Wanner, R M; Castor, D; Güthlein, C; Böttger, E C; Springer, B; Jiricny, J (2009). The uracil DNA glycosylase UdgB of Mycobacterium smegmatis protects the organism from the mutagenic effects of cytosine and adenine deamination. Journal of Bacteriology, 191(20):6312-6319.
Wanner, R M; Güthlein, C; Springer, B; Böttger, E C; Ackermann, M (2008). Stabilization of the genome of the mismatch repair deficient Mycobacterium tuberculosis by context-dependent codon choice. BMC Genomics, 9:249.
Güthlein, C; Wanner, R M; Sander, P; Böttger, E C; Springer, B (2008). A mycobacterial smc null mutant is proficient in DNA repair and long-term survival. Journal of Bacteriology, 190(1):452-456.