Publication: Periapical fluid RANKL and IL-8 are differentially regulated in pulpitis and apical periodontitis
Periapical fluid RANKL and IL-8 are differentially regulated in pulpitis and apical periodontitis
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Rechenberg, D.-K., Bostanci, N., Zehnder, M., & Belibasakis, G. N. (2014). Periapical fluid RANKL and IL-8 are differentially regulated in pulpitis and apical periodontitis. Cytokine, 69(1), 116–119. https://doi.org/10.1016/j.cyto.2014.05.014
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The dental pulp space can become infected due to a breach in the surrounding hard tissues. This leads to inflammation of the pulp (pulpitis), soft tissue breakdown, and finally to bone loss around the root apex (apical periodontitis). The succession of the molecular events leading to apical periodontitis is currently not known. The main inflammatory mediator associated with neutrophil chemotaxis is interleukin-8 (IL-8), and with bone resorption the dyad of receptor activator of NF-κB ligand (RANKL) and osteoprotegerin (OPG). The level
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- Rechenberg, Dan-K
- Bostanci, Nagihan
- Zehnder, Matthias
- Belibasakis, Georgios N
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Rechenberg, D.-K., Bostanci, N., Zehnder, M., & Belibasakis, G. N. (2014). Periapical fluid RANKL and IL-8 are differentially regulated in pulpitis and apical periodontitis. Cytokine, 69(1), 116–119. https://doi.org/10.1016/j.cyto.2014.05.014
